quarta-feira, 30 de julho de 2025

No, Piers. It was a genocide in Gaza from day one. Here's why



Jul 25, 2025

There are too many pundits, like the interminably dim-witted Piers Morgan, who are slowly, oh-so-slowly coming round to the idea that Israel might be committing a genocide in Gaza. But of course, they are still dismissing as “antisemites” those of us who pointed out from the start that it was a genocide.

They hope to get away with this face-saving ploy only because the establishment media continues to ignore what happened before 7 October 2023, events that over many years had made clear Israel was readying to commit genocide – and would grasp a pretext when it arrived.

Here is a brief outline of some of the most pertinent factors:

1. In early 2008 – that is, 17 years ago – deputy defence minister Matan Vilnai, a former senior Israeli general, threatened that Gaza would face a "Shoah" – a word until then, strictly reserved for the Holocaust.

2. He did so shortly after Israel implemented what would become a near two-decade siege of Gaza. Israel had already surrounded the enclave with a heavily militarised fence, made its territorial waters off-limits, and bombed its only airport. From then on, food was tightly rationed, or what Israeli leaders called “putting Gaza on a diet”, while swaths of the enclave were intermittently destroyed by Israeli bombing, or what Israeli leaders called “mowing the lawn”. Gaza was effectively turned into a concentration camp.

3. The siege was complemented by Israel's gradual destruction of Gaza's means of self-sufficiency: any fishing off its coast was stopped; Israel regularly sprayed herbicide on the enclave’s agricultural land; Israel eradicated Gaza's industrial sector by making exports almost impossible; and Israel regularly bombed Gaza's electricity and desalination plants, limiting the essentials of water and power.

4. The goal was clear: to make Gaza entirely dependent on Israel's goodwill, of which there was almost none, and at the same time utterly dependent on aid. In tandem, Israel started waging a deceitful campaign claiming UN aid organisations were linked to Hamas "terror" in the hope it could use this as a rationalisation for impeding aid, as it has done with great ferocity since 7 October 2023, and ultimately for taking over for itself all aid provision, as it has also managed to do in recent months with the creation of an Israeli-US front group, the “Gaza Humanitarian Foundation”.

5. With this as the background, the United Nations warned a decade ago that Gaza was likely to become uninhabitable by 2020. That was a major reason why Palestinians began mass protests at their concentration camp fence in 2018, which Israel responded to with lethal live fire. In one article in the Israeli media at the time, IDF snipers boasted about shooting “42 knees in one day”. Hundreds were killed and many thousands crippled as a result. Those same snipers are currently shooting children in the head, abdomen and testicles, as British surgeon Nick Maynard, who is volunteering in Gaza, has warned.

Let us note too that Israel’s almost complete, and malevolent, control over Gaza – and the fact that the world had lost interest in the enclave’s desperate plight – was a major factor in Hamas and other groups launching their lethal break-out on 7 October 2023.

5. In parallel to all this, and starting in 2007, Israel persuaded the US to join it in a pressure campaign on Egypt: to open its single, short border with Gaza so that the enclave’s people would flood into Sinai – an act of ethnic cleansing and a blatant violation of international law. Egypt refused to submit before 7 October 2023, and has continued to do so since.

In fact, forcibly removing a group from their homes through violence and by making life impossible for them where they live itself meets the legal definition of genocide – all the more obviously so if those doing the forcible removal say that is what they are doing, as Israeli leaders have been stating from the start of their genocidal slaughter and starvation campaign in Gaza.

Israel is committing a genocide to force Egypt and the Arab world to take the people of Gaza as refugees. If they refuse, Israel will continue with the genocide by killing more of Gaza’s people. If they relent, Israel will continue the genocide by dispersing what’s left of the people of Gaza to the far corners of the world. Either way, it is genocide. Either way, it must be stopped – now.

It is cold comfort indeed that, in the very final stages of Israel’s genocide, media pundits like Piers Morgan are ready to concede that a genocide may be about to happen. None of that should obscure or excuse their 21 months of complicity in the genocide that unfolded before all our eyes. They did not know because they did not want to know.

Responsibility for every dead child in Gaza, every maimed child in Gaza, every orphaned child in Gaza, every starving child in Gaza, irreversibly damaged by malnutrition, rests firmly on their shoulders.

 

Whistleblower: IDF & American guards shooting starving Palestinians:  

Source: https://jonathancook.substack.com/p/no-piers-it-was-a-genocide-in-gaza?utm_source=substack&utm_medium=email 

segunda-feira, 28 de julho de 2025

Here's why you should care about Egypt



Kevork Almassian
July 29th

Egypt is under immense pressure from Muslim Brotherhood forces, because the only remaining strong Arab army in the region and the last major obstacle to the Israeli expansionist plan in the Sinai is the Egyptian army.  

Let’s talk honestly—and yes, bluntly—about the Muslim Brotherhood and the broader regional game that’s unfolding around them. People keep asking, “Is the Muslim Brotherhood a terrorist organization?” My answer is yes. Unequivocally. 

But at the same time, we can’t ignore that they’ve been instrumental in various movements throughout the region, especially in the aftermath of the Arab Spring. They’ve been used—let’s not mince words—by global and regional powers alike as tools of influence. 

The last real domino of the Arab Spring was Syria. And once that domino fell, a whole new phase began. Now we’re watching a new narrative unfold around Egypt—specifically, around Gaza. People are throwing accusations left and right: “Egypt is strangling Gaza. Egypt is sealing the Rafah crossing.”

Sure. But have you looked at the map recently? Israel controls the Rafah crossing from the Palestinian side.Even if Egypt opens the gates and pours aid in, it’s Israel that sets the actual limit. So let’s be very clear here: the blockade is enforced with Israeli boots.

But what’s really going on is deeper than border logistics. Egypt is under immense pressure because the only remaining strong Arab army in the region is the Egyptian army. That’s it. Everyone else has been either dismantled, neutralized, or turned into proxy forces for someone else’s agenda. 

We can critique Sisi all day, and I’ll join you, but let’s not throw out strategic reality while we’re at it: the Egyptian military is the last major obstacle to the Israeli expansionist plan in the Sinai, aka the “Greater Israel” project. 

And what’s the easiest way to neutralize that last obstacle? Civil war. Internal chaos. Destabilization from within. 

Enter: the Muslim Brotherhood. Again. 

We’ve seen this movie before. The Brotherhood is already being positioned to ignite unrest. Al Jazeera, for example, is running Egypt-focused coverage 24/7, painting Egypt as the villain starving the Palestinian people. It’s relentless.

And on the ground, we see performative activism that borders on satire: people tossing rice cans into the Mediterranean as if they’ll float to Gaza. NGO cosplay. It’s symbolic warfare designed to sway opinion and destabilize the one Arab country left with military teeth.

If the Brotherhood and their backers really want to help Gaza, they know where the fight is. They know the actual path. But no, that’s not their priority. The same group that sent hundreds of fighters to Syria—Egyptians, Palestinians, and others—now act as if Palestine isn’t worthy of jihad. As if jihad only applies when it’s against other Muslims, against Arab secular regimes. Suddenly, there’s no “energy” for jihad when it comes to facing Israel or defending Gaza. And let me be crystal clear: I am not calling for jihad against Israel or anyone in the region. I am simply showing the ideological farce behind these jihadi movements.

These Takfiri groups in the region have shown their cards. They don’t mobilize for oppressed people unless the oppressor is a convenient target, a politically sanctioned enemy, or part of a broader foreign agenda. It is a mercenary ideology dressed up as religion.

What are we left with? We’re left with tens of thousands of fighters in Syria, many of whom now say they’re tired of war and want peace with Israel. They want peace. And then what do they do? They go and attack defenseless minorities.

So you have enough energy to fight the powerless, but no strength left to challenge actual power? No appetite to confront the real siege around Gaza?

This tells you everything about the nature of the game being played. These aren’t independent actors moved by moral conviction. They’re chess pieces. And someone else is moving them.

If Egypt collapses, what comes next? Who fills the vacuum? It won’t be freedom, it won’t be democracy, and it sure won’t be justice for the Palestinians. It will be chaos. It will be militias. It will be external powers carving up what’s left.

So yes, criticize Egypt where it deserves criticism. But don’t fall into the trap of thinking weakening Egypt strengthens Gaza. That’s the illusion being sold.

                                                           *** 

Subscribe to my YouTube channel for long-form geopolitical analysis and podcast conversations:  https://www.youtube.com/@SyrianaAnalysis

 


Source: https://x.com/KevorkAlmassian/status/1949938207048798302 

Presumed Pathogens and Toxic Truths: Rethinking the Nipah Narrative in Kerala and Beyond

 



Virology Watch

July 26th 2025

🔍 Introduction

In July 2025, two deaths in Kerala were swiftly blamed on Nipah virus. Officials cited PCR detection, activated biohazard protocols, and redirected public fear toward fruit bats—again. But beyond the headlines lies a troubling tendency: illness framed as contagion while toxicological and ecological triggers remain ignored.

This isn’t a novel outbreak—it’s a recycled narrative, where presumed pathogens replace proven causes, and degraded ecosystems escape scrutiny.


🌫️ Kerala 2025: Environmental Breakdown Before Diagnosis

In the weeks leading to the Nipah-linked fatalities, Kerala experienced:

  • The MSC ELSA-3 shipwreck, spilling plastic nurdles, furnace oil, and calcium carbide into coastal waters

  • A BPCL refinery fire (July 8) releasing toxic plumes over populated districts

  • A quarry collapse (June 7) disturbing aquifers and sending particulates into the air

  • The Wan Hai vessel explosion (June 9) leaking unknown chemicals into marine systems

These events overlapped geographically and temporally with reported deaths. Yet no toxicological autopsies were performed. No environmental sampling informed diagnosis. Instead, authorities defaulted to PCR. The narrative took flight—without examining the ground.


🧬 PCR: Sensitivity Without Causality

PCR detects tiny RNA fragments, not intact or infectious viruses. Its limitations are profound:

  • No ability to isolate viable pathogens

  • No demonstration of disease causation

  • Vulnerable to contamination and environmental noise

In polluted environments, RNA fragments may stem from decaying cells, environmental debris, or chemical stress—not viral activity. PCR signals presence, not causality.


🧪 Genome Assembly: Computed Constructs, Not Purified Evidence

What’s called a “Nipah genome” is not the isolation of a whole virus—but the digital stitching of fragmented sequences found in impure laboratory cultures. This process:

  • Relies on computer algorithms to assemble short, non-contiguous RNA sequences

  • Depends on reference genomes, which presume the very pathogen they're trying to confirm

  • Never produces a full, intact, infectious Nipah particle confirmed in vivo

These assemblies are theoretical models—highly suggestive but not experimentally verified. Once submitted to public databases, they gain institutional credibility without ever being subjected to rigorous validation through isolation, replication, or direct causal testing. It’s computational inference masquerading as biological certainty.


🧫 Replication and Cytopathic Effects: Misinterpreted Signals

Claims of viral replication often rest on cytopathic effects (CPE)—changes in cultured cells like syncytia or cell death. But such effects:

  • Can stem from serum additives, mechanical damage, or antibiotic toxicity

  • Occur without purified viral agents

  • Have never been conclusively linked to infectious Nipah particles in vivo

What’s called “replication” may simply be stress response in manipulated lab conditions. Science requires isolation—not inference.


🧪 Antibody Testing: Indirect Inference Without Isolated Antigen

Antibody testing for Nipah hinges on the assumption that synthetic or recombinant proteins represent the real viral antigen. But without isolating the virus itself:

  • The origin of the immune response remains speculative

  • Cross-reactivity with environmental proteins is probable, as is antibody binding to endogenous proteins from damaged tissue, commensal microbes, and components introduced through vaccination.

  • Seropositivity reflects exposure to something—but does not prove infection by Nipah

It’s a diagnostic mirror reflecting shadows, not substance. Positivity becomes interpretation, not proof. This method reinforces the presumed pathogen narrative without ever confirming its physical existence.


🐖 The Pig Culling Campaign: Presumption in Practice

During the 1998–1999 Malaysian outbreak:

  • Over 1 million pigs were culled based on PCR detection

  • No Nipah virus was isolated in vivo from pigs

  • No toxicological examination of farm environments took place—despite heavy use of orchard chemicals and feed additives

Cytopathic changes in pig tissue were interpreted as replication, without appropriate controls. Toxic stress—not presumed viral infection—remains a plausible explanation.


🦇 Defending the Bats

Fruit bats (Pteropus spp.) are repeatedly cast as pandemic scapegoats. Yet:

  • No study has proven transmission of isolated Nipah particles from bats to other species

  • Bats often exhibit RNA fragments and immune markers linked to environmental stress

  • Deforestation and urbanization force bats into human proximity—then demonize them for being there

Bats are not vectors of disease. They are ecological barometers—signaling collapse we choose not to face.


📜 Historical Nipah Outbreaks and Environmental Correlation

Location Year(s) Deaths Environmental Context
🇲🇾 Malaysia 1998–1999 105 Haze crisis, drought, agrochemical use
🇧🇩 Bangladesh 2001–2012 Various Arsenic water, pesticides, unexamined methanol
🇮🇳 India 2001, 2018–2025 Various Sanitation failures, quarry collapses, waste fires
🇵🇭 Philippines 2014 10+ Mining runoff, contaminated forage, no isolation

In each case, environmental degradation mirrored outbreaks. Yet investigation focused on presumed pathogens—while ecological culpability remained untouched.


📊 Patterns and Incentives

Across outbreaks:

  • Diagnostics favor molecular fragments over causal proof

  • Environmental and chemical exposures go unexamined

  • Animals are sacrificed; industrial actors remain shielded

Who gains?

  • Pharma firms secure vaccines

  • Governments evade environmental liability

  • Media amplifies fear

  • Institutions reinforce molecular authority

Sickness is reframed for convenience—not truth.


🧠 Conclusion: Illness, Evidence, and Ecological Reality

Kerala 2025 shouldn't be remembered as another viral episode. It should mark the moment we asked harder questions.

When RNA fragments replace isolated agents, when cell stress mimics replication, and when bats and pigs absorb blame for human negligence—we lose science and gain narrative engineering.


Post-Conclusion Supplement: The Unseen Causality

Ignored Catalysts of Disease

Chronic ecological degradation is a persistent issue across India. Deforestation and habitat loss destabilize ecosystems and increase human exposure to pollutants. Water contamination affects over 70% of surface water, with rivers saturated in effluent, heavy metals, and pharmaceutical waste. Air pollution blankets urban and rural areas alike, with PM2.5 exposure linked to respiratory, cardiovascular, and neurological disorders. Soil toxicity and agrochemical runoff also compromise groundwater, with chronic exposure effects often mirroring infectious syndromes.

Industrial and accidental toxin releases are another neglected dimension. India has witnessed over 130 major chemical accidents in the past decade. While the Bhopal disaster of 1984 stands out historically, smaller-scale releases of neurotoxins, carcinogens, and heavy metals occur regularly with minimal oversight. These events often align with mysterious illnesses, especially in densely industrialized zones and marginalized communities.

Consider the 2025 outbreak in Rajouri, Jammu & Kashmir. A cluster of fatalities drew viral suspicion, yet no infectious agent was found. Instead, cadmium and neurotoxins were detected. Victims showed brain swelling and signs of toxic encephalopathy. Despite toxicological evidence, media and institutions clung to the viral narrative.

The Real Misdirection

These patterns are not fringe phenomena. They are systemic and recurrent. Still, mainstream public health continues to prioritize viral explanations. This results in misdirected interventions—mass culling of livestock, vaccine campaigns, and emergency measures that ignore root causes. Regulatory scrutiny is deflected away from industry and infrastructure, while ecological trauma is medicalized and obscured.

The Missed Opportunity

What appears as infection is instead evidence of environmental collapse. Here lies an opportunity—not for presumed pathogen detection, but for renewed accountability and ecological insight. It’s a chance to restructure diagnostics to integrate toxicological and geospatial analysis, and to demand justice for communities bearing the brunt of industrial negligence.

Reframing the Narrative: “To understand modern epidemics, look not to the genome, but to the geosphere. The virus is a metaphor for mismanagement.”


Source: https://www.reddit.com/r/VirologyWatch/comments/1ma785l/presumed_pathogens_and_toxic_truths/

quarta-feira, 23 de julho de 2025

Charges Dropped Against American Doctor Prosecuted for Fake Vaccinations



The U.S. Department of Justice announced this Saturday that all charges against Dr. Moore, accused of issuing false COVID vaccination certificates, have been dropped. He faced up to 35 years in prison for these acts, even though they were committed in accordance with the Hippocratic Oath and the French Health Code. 

Le Point Critique 
12 July 2025 

On July 7, the trial of Dr. Michael Kirk Moore, a 58-year-old plastic surgeon practicing at the Utah Institute for Plastic Surgery, began in the United States. Dr. Moore refused to vaccinate some patients against COVID during the pandemic, following the intense public outcry sparked by this case. Attorney General Pamela Bondi announced on July 12 that all charges had been dropped.

Dr. Moore was being prosecuted by the Department of Justice (DOJ) for having implemented, within his clinic, with the complicity of several co-defendants, a "scheme" to defraud the U.S. government and the Centers for Disease Control and Prevention (CDC) by circumventing the federal COVID vaccination strategy.

The charges alleged by the DOJ included the destruction of over $28,028.50 worth of Pfizer COVID vaccines, the distribution of nearly 2,000 vaccination certificates issued without administering the product, and injecting children with saline solution. For these various crimes, Dr. Moore faced a 35-year prison sentence.

An extraordinary trial amidst judicial relentlessness

Regarding the first count, although the doses were destroyed because they were not administered, Dr. Moore was officially charged with "conspiracy to convert, sell, transfer, and dispose of public property," in this case, $28,028.50 worth of Pfizer COVID vaccines supplied by the government. This was a drop in the ocean, considering the number of doses he himself had to destroy, now estimated by the Wall Street Journal at over 90 million doses.

Dr. Moore was notably accused of personally enriching himself by selling his false certificates, a claim he has always denied. On the contrary, he explained in an online interview that he treated patients free of charge during the pandemic. He also recounted in this interview the harassment he suffered at the hands of the Department of Justice, including the tapping of his phones and his incarceration for 22 days, with 22-hour solitary confinement, for attempting to communicate essential judicial information to his co-defendants.

Regarding the other two charges, the federal government ruled in 2023 that Dr. Moore had endangered "the health and well-being of his patients" by taking advantage of their weakness. However, these false certificates were all issued at the patients' request and solely to allow them to circumvent vaccination mandates, which have now been rescinded or deemed illegal in more than half of the US states. Similarly, the saline injections were all administered with the consent of the parents, who were concerned about protecting their children, for whom the new CDC team had recently removed the COVID-19 vaccine from the vaccination schedule.

A few days before the start of his trial, Dr. Moore recalled in an independent media outlet that the vaccine information leaflet given to healthcare professionals is a bulky pamphlet with the following inscription: "Intentionally left blank." It should be noted that the Hippocratic Oath and the French Public Health Code prohibit a physician from administering a product if they are not certain or firmly convinced that it will not cause harm to the recipient.

The decision to prosecute Dr. Moore in January 2023 was all the more surprising given that Utah passed House Bill 131 in March 2023, which prohibits businesses and public entities from discriminating against employees or the public based on vaccination status. 

An Unexpected Twist During the Trial

This case deeply moved America, which mobilized in a historic way in support of the doctor. He notably received public support last April from the Secretary of the U.S. Department of Health and Human Services (HHS), Robert F. Kennedy Jr. However, the spotlight remained on Attorney General Pamela Bondi, whose ties to Pfizer are currently the subject of controversy.

In a tweet posted this evening on her X account, she finally announced that she had put an end to a completely unfair situation, a few days after the public intervention of Republican Congresswoman Marjorie Taylor Greene, much to the dismay of Le Monde: 

Will this case set a precedent? Probably not, since it was closed based on a discretionary decision by the Department of Justice, without a decision being rendered on the merits. But it does suggest that citizens have understood that they suffered a historic attack on their fundamental rights during the pandemic and that they are now ready to stand with the doctors who refused to obey orders and who are the Schindlers of 2020.

Source:  https://lepointcritique.fr/2025/07/12/abandon-charges-contre-medecin-americain-poursuivi-pour-fausses-vaccinations/

Terrain



Virology Watch
July 7th

Terrain is a powerful word—especially in biology, where it doesn’t refer to landscape or structure, but to the living field through which coherence arises: where cells communicate, bacteria collaborate, and tissues align with their surroundings.

Within the body, terrain forms an internal environment—a living matrix of cells, bacteria, fluids, and signals. It is shaped by what surrounds it and reflects what it receives. When the terrain is coherent, it expresses health. When it is burdened beyond its capacity, it expresses dysfunction. Disease, then, is not an invasion by so-called pathogens, but a signal of imbalance. To understand terrain is to understand the conditions under which life maintains its form—and the thresholds beyond which it begins to unravel.

The state of the terrain reveals the body’s trajectory. Patterns of vitality or dysfunction emerge not from pathogenic invasion, but from accumulated responses to environmental conditions. Coherence marks the system’s capacity to integrate change; imbalance signals its thresholds have been exceeded. Health, then, is not enforced by interventions—it is witnessed in the terrain’s ongoing ability to sustain its own integrity under external influence.

The Illusion of Invasion: Germ Theory and the Myth of the Siege

In sharp contrast to the relational coherence of terrain, germ theory frames the human organism as a citadel—an isolated entity under perpetual threat from the outside. It envisions disease not as a dysfunction in sustenance or coherence, but as a result of external attack by independent, invasive microbes. This model doesn’t just propose treatment; it demands defense. Every cough becomes a signal of war. Every immune response becomes a battlefield report.

But this narrative is not born of nature. It is born of distortion in the human psyche—a projection of fear, a misinterpretation of relation, a craving for control.

Where terrain theory sees the organism functioning in context, germ theory isolates, imagines siege, and then retroactively builds evidence to justify its assumptions. It redefines disease as invasion and health as surveillance, generating entire industries dedicated to sterilization, vaccination, and medical preemption.

The result is not safety, but addiction to defense—to inoculation, to prophylaxis, to purification. This is not medicine. It is a system of control masquerading as care.

The Demand for Purity: Proxy Logic and the Weaponization of Care

At the root of germ theory lies a hidden logic: the need for purity. It does not account for terrain degradation caused by environmental toxicity, social impoverishment, or emotional trauma. Instead, it invents a culprit—the pathogen—a stand-in for all complexity.

This is a form of proxy logic: the substitution of imagined causes for real conditions. And once this proxy is accepted, the interventions it legitimizes take root in the body as law:

  • The pathogen becomes enemy

  • The immune system becomes a security apparatus

  • The doctor becomes a commander

  • The body becomes occupied territory

This response pattern is not accidental, but neither is it necessarily malicious. It is learned behavior—an inherited, intuitive strategy rooted in fear, projection, and the desire for certainty. It represents a misguided intuition: the belief that threats must be simple, visible, external. And so systems of care transform into systems of command—not because life demands it, but because the logic of control has been taught, rehearsed, and institutionalized.

In this model, fear is not merely a symptom. It is a way of knowing. And once that way takes hold, obedience becomes instinct—and truth, the casualty.

The Trojan Horse: Entrapment by Means of Protection

The architecture of germ theory is a Trojan horse—a strategy of entrance through deception. Appealing to the desire for protection, it infiltrates the gates of thought, rewriting how life is understood. It was not fear that breached the gates—it was the theory that rewrote life as siege. Once inside, it rewires the organism’s relationship to itself. No longer is terrain sustained by alignment. It is policed by vigilance. The environment is no longer a condition to be honored, but a threat to be sanitized. The body is no longer the living soul, but a potential biohazard.

Health becomes a theater of war.

And in this system, the constant escalation of intervention is not an unfortunate consequence—it is the measure of success. Each new pathogen justifies more surveillance, more compliance, more surrender of sovereignty over one’s own terrain. The system doesn’t just respond to threat. It requires it.

Toward Restoration: Reclaiming Meaning, Reframing Bacteria

If the siege is illusion, then the task is not to fight but to sustain. The terrain possesses a conditional capacity for repair—activated through its own function—but only when the surrounding environment provides the necessary coherence. Restoration begins not through external force, but from the terrain’s own integrative response—provided it is not overwhelmed by industrial toxicants, nutritional imbalances, unresolved emotional trauma, or the unnecessary imposition of pharmaceutical agents.

This is where misinterpretation becomes destruction. In moments of imbalance, bacteria—typically viewed as beneficial or neutral—often rise to support repair: breaking down damaged material, buffering toxins, or restoring metabolic function. But when this activity is mistaken for aggression, germ theory intervenes. It labels helpers as culprits, sends in antibiotics, and disrupts the very agents of coherence. The result is not healing, but escalation.

Consider: the house is on fire. The fire brigade arrives. But before they can douse the flames, someone mistakes their tools for weapons and arrests them. Now the fire spreads. Not because of neglect—but because meaning was lost.

This is what germ theory does when it collapses context. It identifies bacteria as pathogens not because of what they are, but because of when they arrive. Bacteria are not toxins. They are living organisms capable of extraordinary symbiosis—until assaulted. Under direct pressure from pharmaceutical agents or environmental toxins, their function may shift. Some begin producing toxic byproducts—not out of aggression, but as a reaction to being chemically or structurally damaged. The system is not failing; it is under attack. In that altered state, even bacteria that once supported coherence may appear harmful—not by intention, but by consequence.

This distortion of bacterial function is not the end of the error—it is its beginning. Germ theory doesn’t stop at misreading living organisms under duress; it extends that logic beyond biology itself. It projects pathogenic intent onto theoretical entities that do not metabolize, move, or self-replicate: so-called viruses. Unlike bacteria, these viruses are introduced as entities that do not exhibit the relational behavior of life—yet germ theory collapses that boundary too, preserving its invasion script at the cost of coherence.

Viruses are not classified as microbes in terrain theory because they have never been isolated according to the standards of the scientific method. They have not been directly observed as intact, replicating entities under light microscopy, cultured independently, or demonstrated to act in the manner claimed. What is referred to as a virus is a model constructed from fragments—genetic material inferred and assembled by computers into theoretical genomes. No complete, replicating structure has ever been obtained. Assertions about viral behavior are not supported by verified physical specimens. Claims about infection or replication are made absent the object itself. Effectively, terrain theory regards viruses as non-existent.

The same logic applies to bacterial vaccines. Once bacteria are understood not as initiators of disease but as responders to ecological distress, the rationale for vaccinating against them collapses. Such procedures do not address root causes, but instead reinforce a mischaracterization of microbial behavior that terrain theory fundamentally rejects.

To restore health, we must realign meaning. The body does not require warfare against the agents it calls to help. It requires the removal of external pressures—environmental toxins, emotional fragmentation, chemical intrusion—that exceed its capacity to maintain internal order. Healing does not come by destroying the elements that arise in response. It comes by correcting the conditions that forced them to act. In that correction, the terrain does not initiate defense in the classical sense of opposition or attack. It restores through purging, rebalancing, and releasing what no longer serves—not to fight, but to return to function.

Thresholds and Consequences: When Restoration Yields to Compensation

There is a critical distinction between intervention and compensation. Certain pharmaceuticals—when terrain has been irreparably altered—may serve as mechanical aids: not to heal, but to substitute a lost function. Yet even these must be examined rigorously, for their mechanisms often produce effects that extend far beyond their intended purpose. Restoration is not their logic—management is. Vaccination, however, operates differently. It does not compensate for dysfunction; it presupposes invasion. It imposes a narrative of defense where no pathology yet exists. It intervenes not in response to collapse, but in anticipation of one—often by disrupting a terrain that has not called for rescue, causing systemic effects the terrain never requested and may not be equipped to reconcile.

Summary: Respecting the Terrain

The terrain is a responsive system—continually shaped by the quality of air, water, food, human interaction, and stress. Health is its expression when those inputs support coherence rather than disrupt it. When bacterial activity is misread as pathology, interventions often override the body's intelligence instead of listening to what it reveals.

What’s needed is context, not control: the ability to discern when a response signals dysfunction, and when it reflects adaptation to adverse conditions. Pharmaceuticals may assist in cases where function has been lost, but their use must be evaluated with care. Vaccination, by contrast, imposes interference where no failure exists—disrupting a system that remains intact.

Respecting the terrain means allowing its processes to unfold without unnecessary interruption, while actively removing the pressures that compromise its function: chemical exposures, poor nutrition, chronic stress, manipulative health messaging, and institutional practices that prioritize control over understanding. Health emerges not through imposition, but through conditions that allow coherence to sustain itself.


Source: https://www.reddit.com/r/VirologyWatch/comments/1lttfm5/terrain/

terça-feira, 22 de julho de 2025

Breathing Electrons

 



Unbekoming
Dec 25, 2024

This recent paper by Pollack rings true to me. 

 


Put simply, Pollack is making the following points:

Instead of breathing in oxygen gas molecules as we've always thought, Pollack suggests our lungs are actually stealing electrons (electrical charges) from oxygen in the air. Think of oxygen as a delivery truck carrying valuable electrons. When we breathe, our lungs don't take the whole truck (oxygen molecule) - they just unload the cargo (electrons).

These electrons are then picked up by our red blood cells, which act like delivery vans distributing the electrons throughout our body where cells need them for energy. This explains why fish can live in deep water with very little oxygen - they've figured out how to get electrons directly from water instead of air.

It's a bit like the difference between having to transport gasoline (oxygen molecules) versus just transmitting electricity (electrons). Pollack thinks our bodies work more like electrical systems than chemical ones, making breathing a way to harvest electricity rather than collect gas.

This obviously makes me think about childhood vaccines.

Childhood vaccines often include small amounts of aluminum compounds as adjuvants, which “help” the immune system mount a stronger response. From a bioenergetic perspective, aluminum can bind to red blood cells (RBCs) and neutralize some of the negative charge they need to repel each other and efficiently transport electrons. Disrupting this charge balance may slow blood flow and reduce the delivery of both electrons and oxygen to tissues.

Aluminum can also bind with enzymes and proteins, leading to potential changes in how RBCs manage electron transfer. Over time, this electropositive influence can place extra strain on detoxification pathways and increase the overall demand for electrons in the body. Research on aluminum’s accumulation and toxicity (Shaw CA, Seneff S, et al. “Aluminum in the central nervous system (CNS): Toxicity in humans and animals, vaccine adjuvants, and autoimmunity.” Immunologic Research (2013)) underpins concerns that even small amounts, when repeatedly introduced, may disrupt normal bioenergetic processes.

With thanks to Gerald Pollack.

Analogy

Think of your body as a massive collection of tiny electronic devices, similar to cell phones that need constant charging. In the traditional view, we thought of breathing like filling up gas tanks with oxygen - moving actual gas molecules around the body to power these devices.

However, this new theory suggests that breathing works more like a wireless charging system. Just as your phone doesn't need the actual electricity from the power plant to flow into it (it just needs the energy transfer), your body might not need actual oxygen molecules to travel through it. Instead, the lungs act like a charging station, extracting electrons (electrical charge) from oxygen molecules in the air. Your red blood cells work like little battery packs, collecting these electrons in the lungs and distributing them throughout your body where needed.

This explains why fish can survive in deep water with little oxygen - they're essentially using a different "charging adapter" that can pull electrons directly from water instead of air. It's like how your phone can charge from different power sources (wall outlet, car charger, portable battery pack) as long as it gets the electricity it needs.

Just as you don't have to understand the complex physics of electricity to charge your phone, your body doesn't need to move actual oxygen molecules around - it just needs the electrons that oxygen can provide. This new perspective suggests we're not so much air-breathing creatures as we are electron-harvesting beings.

Is it oxygen, or electrons, that our respiratory system delivers?

Gerald Pollack Paper
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12-point summary

  1. Fundamental Challenge to Traditional Model: The conventional understanding of respiratory gas exchange cannot adequately explain why oxygen passes through alveolar membranes while nitrogen, despite being smaller and more abundant, cannot. This paradox suggests our basic understanding of respiration needs revision.

  2. Alternative Mechanism: Rather than transporting oxygen molecules, the respiratory system may primarily function to extract electrons from oxygen. This explains the selective nature of gas transport and provides a more direct connection between respiration and cellular energy needs.

  3. Hemoglobin's True Role: Instead of carrying oxygen molecules, hemoglobin may function as an electron carrier, switching between positively charged (electron-accepting) and negatively charged (electron-loaded) states. This explains its color changes and oxidation tendencies.

  4. Capillary Design Purpose: The seemingly inefficient design of capillaries being narrower than red blood cells serves to ensure tight contact for electron transfer, while their sparse distribution around alveoli is sufficient for electrical rather than gas transfer needs.

  5. Deep-Sea Evidence: The survival of deep-sea fish in oxygen-poor environments through electron extraction from water, rather than oxygen usage, provides compelling evidence that electron transfer, not oxygen transport, is fundamental to respiration.

  6. Seawater Experiments: Quinton's dramatic experiments showing survival after blood replacement with seawater suggest that electron availability, rather than oxygen-carrying capacity, is crucial for maintaining life.

  7. Blood Substitute Insight: The effectiveness of perfluorocarbon blood substitutes can be explained by their high electronegativity rather than oxygen-carrying capacity, supporting the electron transfer model.

  8. Cellular Water Structure: The discovery of EZ (exclusion zone) water in cells provides a mechanism for storing and utilizing electrons, creating a direct link between respiratory electron delivery and cellular function.

  9. Phase Transitions: Cellular work is powered by phase transitions involving electron movement, suggesting a direct connection between respiratory electron delivery and cellular activity.

  10. Natural Examples: Fish gill function and the presence of nitric oxide in exhaled breath provide natural evidence of electron-based respiratory processes.

  11. Experimental Validation: The theory can be tested through specific experiments measuring electrical charges in expired air and examining plasma oxygen content, offering clear ways to validate or refute the hypothesis.

  12. Broader Implications: This model suggests biological systems function primarily as electrical rather than chemical machines, potentially revolutionizing our understanding of physiology and medical treatment approaches.

Some thoughts

Gerald Pollack’s theory suggests that atmospheric oxygen serves as a direct source of negatively charged electrons for red blood cells (RBCs). In this view, each oxygen molecule donates two or more electrons, which RBCs then transport throughout the body. The oxygen itself remains in the lungs, and the oxygen exhaled carries a more positive charge than what was inhaled. This perspective ties into bioenergetic medicine by emphasizing that our bodies are continuously harvesting electrons not just from inhaled oxygen, but also through the skin (grounding) and intestines (food). The goal is to maintain a balanced negative charge (approximately -25 millivolts, or a pH near 7.44) to support optimal cellular function.

A core element of Pollack’s idea is that RBCs require a strong negative charge to repel from endothelial surfaces, ensuring efficient blood flow. When carbon monoxide donates only one electron, it may neutralize RBCs instead of fully charging them. The result is a collapse of this zeta potential and a buildup of traffic in capillaries, potentially explaining why carbon monoxide is so deadly. If alveoli become clogged or lose function, the continuous supply of electrons from the lungs would diminish, weakening overall energy delivery to cells.

Lung capacity emerges as a primary indicator of overall health by governing how much negative charge RBCs can acquire and transport. As lung function declines, so does the body’s total energy supply, accelerating chronic conditions. There are proposals that an implanted electrode could supplement the body’s negative charge, bypassing the lungs if necessary and theoretically preserving blood pH. This technology, if successful, might reduce dependence on oxygen intake and even open the door to underwater respiration without scuba gear.

Grounding, structured water, and bioenergetic devices (such as Tennant’s Transducer) all tie into this concept of restoring or maintaining adequate negative charge. Pollack’s work resonates with earlier ideas (such as Tilden’s notion of “nerve energy”) by reframing much of biology and medicine around the simple yet powerful concept of electron flow. Sources that explore these concepts include Gerald Pollack’s investigations into water structure (Pollack GH. The Fourth Phase of Water: Beyond Solid, Liquid, and Vapor. Ebner & Sons, 2013) and Dr. Jerry Tennant’s discussions on voltage in healing. This perspective champions a bioenergetic foundation of health and suggests that maintaining strong electron flow is critical for longevity and vitality.

25 Questions & Answers

Question 1: Why does the traditional understanding of respiratory gas exchange face challenges in explaining selective gas passage?

The conventional model struggles to explain why oxygen can pass through alveolar membranes while nitrogen, despite being smaller and more abundant, cannot. This selective passage presents a paradox, as certain toxic gases larger than nitrogen, such as fluorine and chlorine, can also pass through these membranes, indicating that molecular size alone cannot explain the selective nature of gas transport.

The diffusion-based explanation faces further complications when considering that gases form bubbles in liquids, raising questions about how these bubbles could pass through continuous membranes. The dissolution of oxygen in membrane water has been proposed as an alternative, but the extremely low solubility of oxygen in water (approximately 10 molecules per million) makes this mechanism quantitatively insufficient to support life.

Question 2: How does the electron transfer hypothesis differ fundamentally from the conventional oxygen-based respiration model?

Instead of whole oxygen molecules passing through alveolar membranes, the electron transfer hypothesis proposes that only electrons extracted from oxygen molecules make the journey. These electrons are then transported by hemoglobin through the bloodstream directly to tissues where they support metabolism. This mechanism eliminates the need to explain how gas molecules physically traverse membrane barriers.

The proposed model suggests that oxygen serves primarily as an electron donor rather than a respiratory gas. This fundamental shift in understanding transforms the respiratory system from a gas exchange mechanism to an electron harvesting system, where oxygen's high electronegativity makes it an ideal electron source, but not the essential component itself.

Question 3: What role does electrical charge play in the proposed new model of respiration?

Electrical charge serves as the primary driver of respiratory function, with oxygen's high electronegativity making it an excellent electron donor. The model suggests that positively charged hemoglobin draws electrons from oxygen molecules across the alveolar-capillary interface, creating a direct electrical transfer rather than a gas exchange process.

This electrical mechanism explains various observed phenomena, including the selective nature of gas passage and the requirement for tight contact between red blood cells and capillary walls. The transfer of electrons rather than molecules provides a more streamlined explanation for how respiratory products reach their cellular destinations and support metabolic processes.

Question 4: Why can't nitrogen pass through alveolar membranes despite being smaller than oxygen?

The inability of nitrogen to pass through alveolar membranes, despite its smaller size and greater atmospheric abundance, challenges traditional size-based diffusion models. In the electron transfer hypothesis, this selectivity makes sense because nitrogen lacks oxygen's strong electronegativity and thus cannot participate in the electron donation process that characterizes true respiratory function.

The fact that nitrogen remains excluded even during deep breathing, when alveolar pores would presumably be more open, further supports an electron-based rather than size-based selection mechanism. This observation aligns with the idea that respiratory gas selection depends on electrical properties rather than physical characteristics.

Question 5: How does the proposed electron-based mechanism explain the passage of toxic gases?

The electron-based mechanism explains toxic gas passage through the alveolar membrane based on the gases' electrical properties rather than their size. Halogens like fluorine and chlorine, despite being larger than nitrogen, can pass through because they interact with the electron transfer system, potentially disrupting normal electron flow patterns.

This electrical interaction explanation provides a more consistent model for understanding why certain gases can traverse the alveolar barrier while others cannot. Rather than relying on physical characteristics like molecular size, the model suggests that a gas's ability to participate in electron transfer determines its passage through respiratory membranes.

Question 6: What are the two distinct states of hemoglobin and how do they relate to electrical charge?

Hemoglobin exists in two well-documented forms: the T form, associated with low pH and positive charge, and the R form, associated with high pH and negative charge. These states correlate with hemoglobin's ability to first attract electrons from oxygen and then deliver them to tissues, creating a cycle of electron acceptance and donation.

These distinct charge states enable hemoglobin to function as an electron carrier rather than merely an oxygen transporter. The positive T form attracts electrons from oxygen at the alveolar interface, while the resulting R form carries these electrons through the bloodstream until they can be delivered to tissues, whereupon the molecule returns to its positive T state.

Question 7: Why are capillaries narrower than red blood cells, and how does this support the electron transfer theory?

Capillaries in healthy young adults measure only 3-4 micrometers in diameter, notably smaller than the 6-7 micrometer diameter of red blood cells. This forced squeeze ensures intimate contact between red blood cells and capillary walls, creating optimal conditions for electron transfer across the alveolar-capillary interface.

The energy expenditure required to force red blood cells through these narrow vessels, rather than being an inefficient design, serves a crucial purpose in the electron transfer model. The tight contact eliminates potential insulating gaps and maximizes electrical conductance, particularly through the highly conductive surfactant layer lining the alveolus.

Question 8: How does hemoglobin's oxidation tendency support the electron transfer hypothesis?

Hemoglobin's natural tendency to oxidize, often considered problematic for blood storage, actually supports its proposed role as an electron carrier. This oxidation tendency demonstrates hemoglobin's ability to readily release electrons, a crucial characteristic for delivering electrons to tissues after acquiring them from oxygen at the alveolar interface.

The preference for losing electrons in pairs rather than singly aligns with the observation that toxic electron-donating gases like hydrogen sulfide cannot substitute for oxygen. This suggests that the respiratory process requires the transfer of multiple electrons simultaneously, a capability that oxygen possesses but single-electron donors lack.

Question 9: What explains the color difference between arterial and venous blood in the electron model?

The distinctive color difference between red arterial blood and purple venous blood reflects hemoglobin's different charge states rather than oxygen content. The color change corresponds to hemoglobin's transition between its electron-rich and electron-depleted states as it performs its electron transport function.

This interpretation challenges the traditional explanation of color differences being due to oxygen saturation levels. Instead, it suggests that the color changes directly reflect hemoglobin's electrical state, providing visible evidence of its role in electron transport rather than gas transport.

Question 10: Why is the sparseness of capillaries around alveoli significant to the proposed theory?

The relatively sparse distribution of capillaries around alveoli, which seems inefficient for gas exchange, makes more sense in an electron transfer model. Since electron transfer can occur more efficiently than gas diffusion, fewer points of contact between capillaries and alveoli are needed to achieve adequate electron harvesting from oxygen.

This apparent design limitation in the traditional gas exchange model becomes a logical feature in the electron transfer hypothesis. The arrangement provides sufficient electrical contact points while minimizing the energy cost of maintaining extensive capillary networks, suggesting an optimized rather than compromised design.

Question 11: How do deep-sea fish survive with limited oxygen, and what does this suggest about respiration?

Deep-sea fish thrive in oxygen-poor environments by extracting electrons directly from water rather than relying on dissolved oxygen. When water passes through their gills, it becomes more acidic (positively charged) upon exit, indicating that the gills have extracted negative charges (electrons) from the water molecules.

This mechanism explains why fish cannot survive in air despite abundant oxygen - they lack the machinery to extract electrons from oxygen gas, having evolved to obtain electrons from water instead. This alternative electron-harvesting strategy supports the broader hypothesis that respiration fundamentally involves electron transfer rather than oxygen transport.

Question 12: What significance do Quinton's seawater experiments have for the electron transfer theory?

Quinton's experiments demonstrated that dogs could survive after having their blood replaced with seawater, despite the dramatic reduction in hemoglobin concentration. These dramatic findings suggest that the critical respiratory component isn't oxygen-carrying capacity but rather the ability to deliver electrons to tissues, which the negatively charged components of seawater could provide.

The dogs' recovery from near-death states following seawater infusion indicates that traditional oxygen transport via hemoglobin may not be as essential as previously thought. Instead, the electrically conductive properties of seawater might have provided an alternative electron delivery system to sustain cellular function.

Question 13: Why do perfluorocarbons work effectively as blood substitutes according to the new theory?

Perfluorocarbons' effectiveness as blood substitutes can be explained by their high electronegativity, which exceeds even that of oxygen. Their strong electron-attracting properties make them capable of serving as electron carriers in the absence of hemoglobin, suggesting that their success isn't due to oxygen-carrying capacity but rather their ability to participate in electron transport.

This interpretation aligns with the electron transfer hypothesis by demonstrating that substances with strong electron-attracting properties can substitute for hemoglobin's function, regardless of their gas-carrying capabilities. The focus shifts from gas transport to electron management as the critical factor in maintaining tissue viability.

Question 14: How does the fish gill mechanism support the electron transfer hypothesis?

Fish gills demonstrate a clear electrical component to respiration, as evidenced by the pH changes in water passing through them. The exit of more acidic (positively charged) water indicates that gills extract negative charges (electrons) from water molecules, establishing a direct electron harvesting mechanism that operates independently of oxygen.

This gill function provides a natural example of electron-based respiration that doesn't require oxygen gas, supporting the broader hypothesis that electron transfer, rather than oxygen transport, is the fundamental respiratory process. The fact that fish can't survive in air despite abundant oxygen further supports this interpretation.

Question 15: What does exhaled nitric oxide suggest about the respiratory process?

The presence of nitric oxide in exhaled breath suggests that inspired oxygen undergoes electrical changes during respiration. After oxygen molecules donate their electrons, the resulting positively charged oxygen can react with nitrogen to form nitric oxide, providing evidence that oxygen's role involves electron donation rather than simple gas transport.

This observation helps explain what happens to oxygen molecules after their electrons are extracted, completing the picture of the respiratory cycle. The formation of nitric oxide serves as a natural byproduct of the electron extraction process, providing observable evidence of the proposed mechanism.

Question 16: What role does EZ (exclusion zone) water play in cellular function?

EZ water, formed adjacent to hydrophilic surfaces within cells, creates a negative charge separation that contributes to cellular electrical potential. This structured water fills much of the cellular space and maintains a sustained negative electrical potential, providing a reservoir of electrons for cellular functions.

The presence of EZ water supports the electron-based respiratory model by providing a mechanism for storing and utilizing electrons within cells. Its ability to maintain charge separation helps explain how cells can sustain their electrical potential and utilize electron energy for various cellular processes.

Question 17: How does cellular electrical potential relate to the proposed respiratory mechanism?

Cellular electrical potential, maintained by negatively charged EZ water, represents stored electron energy that cells can utilize for various functions. The respiratory system's delivery of electrons helps maintain this potential, creating a direct link between respiration and cellular energy storage.

This electrical system provides a more direct pathway for energy transfer than traditional metabolic models, with electrons from respiration directly supporting cellular electrical potential. The maintenance of this potential becomes a primary function of respiration, linking respiratory electron delivery to cellular energy states.

Question 18: What is the relationship between electron transfer and cellular phase transitions?

Cellular phase transitions, involving the conversion between EZ water and ordinary water states, along with protein conformational changes, represent the expenditure of stored electron energy. These transitions power cellular work, including contraction, secretion, and nerve conduction, directly linking electron availability to cellular function.

The system requires a constant supply of electrons to restore the high-energy state after each transition, creating a direct connection between respiratory electron delivery and cellular work capacity. This mechanism provides a more direct link between respiration and cellular function than traditional metabolic pathways.

Question 19: How does water splitting in cells support the electron-based theory?

Water molecules near hydrophilic surfaces split into positive and negative components, with the negative component forming EZ water. This natural charge separation process provides a mechanism for storing and utilizing electrons delivered by the respiratory system, supporting the idea that electron management is central to cellular function.

Laboratory studies have confirmed that direct electrical current can convert ordinary water to EZ water, demonstrating the feasibility of electron-based water structuring. This provides experimental support for the role of electrons in maintaining cellular water structure and function.

Question 20: Why is the surfactant layer's conductance important in this model?

The high conductance of the alveolar surfactant layer facilitates efficient electron transfer from oxygen to hemoglobin. This conductivity provides a crucial pathway for electron movement across the alveolar-capillary interface, supporting the proposed electron transfer mechanism.

The surfactant's conductive properties, combined with the tight contact between red blood cells and capillary walls, creates optimal conditions for electron extraction from oxygen. This arrangement explains why the respiratory system's structure is optimized for electrical conductance rather than gas diffusion.

Question 21: How can the electron transfer hypothesis be experimentally validated?

The hypothesis can be tested by measuring the electrical charge of expired air to detect positively charged oxygen molecules, providing direct evidence of electron extraction. Additional experiments could examine whether direct electron transfer can convert hemoglobin between its different states, and analyze plasma oxygen content to confirm the absence of molecular oxygen transport.

These proposed experiments focus on detecting electrical changes rather than gas movements, offering clear ways to distinguish between traditional gas transport and electron transfer mechanisms. The results could provide definitive evidence for or against the electron transfer model.

Question 22: What does oximeter function reveal about the proposed mechanism?

While oximeters are commonly thought to measure oxygen saturation, they actually detect structural differences between arterial and venous hemoglobin through light absorption. The device cannot distinguish whether these differences result from oxygen binding or electron transfer, making its measurements compatible with either model.

The oximeter's function therefore doesn't contradict the electron transfer hypothesis, as it simply detects hemoglobin state changes without revealing their underlying cause. This interpretation suggests that common medical devices may be measuring electrical states rather than oxygen levels.

Question 23: Why can't gases like hydrogen sulfide substitute for oxygen despite being electron-donors?

Although gases like hydrogen sulfide can donate electrons, they lack oxygen's capacity to donate multiple electrons simultaneously. Oxygen's multiple oxidation states (-2, -1, 0, +1, +2) allow it to participate in multi-electron transfers, while single-electron donors cannot support the natural two-electron oxidation preference of hemoglobin.

This limitation explains why not all electron-donating gases can support respiration, providing a logical framework for understanding gas toxicity based on electron transfer capabilities rather than traditional gas exchange properties.

Question 24: How does this theory establish a direct link between respiration and metabolism?

The electron transfer theory creates a direct connection between respiratory function and cellular metabolism by showing how electrons move from inspired oxygen directly to cellular components. This direct transfer eliminates the need for complex intermediate steps, providing a more streamlined explanation for how respiratory activity supports cellular function.

The model shows how respiratory electrons directly maintain cellular electrical potential and power phase transitions, creating a clear pathway from respiration to cellular work. This direct connection helps explain the immediate effects of respiratory disruption on cellular function.

Question 25: What broader implications does this theory have for understanding biological systems?

The electron transfer hypothesis suggests that biological systems function primarily as electrical rather than chemical machines, with electron movement serving as the fundamental basis for physiological processes. This perspective unifies various biological phenomena under a common electrical framework, from neural function to cellular metabolism.

This electrical paradigm could lead to new approaches in medical treatment and biological research, focusing on managing electron flow rather than chemical processes. The theory suggests that many biological processes might be better understood and manipulated through their electrical properties rather than their chemical characteristics.

 VIDEO: We Breathe Electricity Not Oxygen - Dr Tom Cowan  

 Source: https://unbekoming.substack.com/p/breathing-electrons