María José MARTINEZ ALBARRACIN
Doctor of Medicine from the University of Murcia, Spain. She is a professor of Clinical Diagnostics, as well as a professor of Biochemistry, Immunology, and Laboratory Instrumental Techniques.
El Correo de España. 24/12/2021
No Más Mentiras comments:
Dr. María José Martinez Albarracin was amongst the earliest members of the Spanish group Médicos por la Verdad (Doctors for Truth) and has been studying, researching, publishing articles and participating in the battle against the Plandemic on social media platforms and in the handful of mass media outlets that have allowed her to express herself publicly. On Christmas Eve, we reproduce this recent and important piece of hers, which clearly sets out something that was almost obvious to anyone applying common sense. The common flu almost disappeared from the moment the Coronavirus Pandemic was declared, because it was "reclassified" as Covid-19, as part of the plan to inflate cases and disseminate fear in society. At the end of 2021, where the farcical mood of the Plandemic is falling more and more to the ridiculous, although no less dangerous in its intentions, Ómicron is a forward flight of globalist designers; the ailment it causes is no longer flu-like but colds. Any old sore throat, headache, cough, or night sweats could be “symptoms” of the elusive and unlikely SarsCov-2!
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When we first started learning about the "new disease" supposedly caused by the new Chinese virus Sars-CoV-2 in the winter of 2019-20, we barely knew that it was a bilateral interstitial pneumonia. Then we learned that there were patients who died from a hyperacute inflammatory syndrome or "Cytokine Storm" and finally, when a group of Italian pathologists began to perform autopsies (ignoring the WHO's recommendations) we learned that the endothelium of various blood vessels, including arteries, were damaged, with microthrombi and accumulations of neutrophil extracellular traps (NETs).
Since then, despite being repeatedly lectured by the ruling party and the mass media that we are supposedly suffering outbreaks and "successive waves" of Covid-19, the clinical details and pathophysiology of the aforementioned disease has no relation to the spring 2020 epidemic, which occurred almost synchronously, in many countries of the old world. During the summer of 2020, we only had colds and asymptomatic positive PCR cases, and in the autumn-winter, some additional pneumonia. But then the Covid vaccines arrived and with them, the "variants" of the virus. The British and South African variants arrived as soon as the inoculations just began. The delta variant (which was no longer suitable to be dubbed with the name of a country) arrived with the second doses and now the omicron coincides with the third dose. None of this is not simple coincidence since, according to the study entitled "Mechanisms of SARS-CoV-2 Evolution Revealing Vaccine-Resistant Mutations in Europe and America" (1) the variants occur precisely in countries with higher rates of Covid vaccination.
In the autumn-winter of 2020, the year in which the flu disappeared throughout Europe, Dr. Schmied, a pathologist at the University of Ulm, used electron microscope photos of the bronchial lavage of Covid patients along with subsequent cell cultures, She found some viral particles, the least in number being coronaviruses, but only in immunosuppressed patients with pulmonary symptoms. She found in particular staphylococci, streptococci, adenovirus and very frequently, Borrelia. And we know that Borrelia appears with immunosuppression.
If Covid-19 were a disease, in the way that is described in medical nosology, all patients in all seasons would present the same clinical picture with very little variation. But since, as we know, there is so much variability in symptoms, Covid-19 is not a new disease but a syndrome that occurs as a complication of the flu (or Covid vaccines) and is characterised by an alteration or dysregulation of innate immunity.
What exactly is this dysregulation?
Until now, it has not been scientifically proven that the Covid syndrome is produced unequivocally or exclusively by the SARS-CoV-2 virus, because it is diagnosed by PCR tests authorised by the emergency means, The package insert of those tests clearly states that they are not suitable for clinical diagnosis and, as we know, they can produce false positive resutls from other viruses related to influenza syndromes.
It is absurd to claim that the same virus can produce clinical profiles as diverse as the absence of symptoms, a mild cold, bilateral interstitial pneumonia and multiorgan vasculitis with an acute and fatal cytokine storm. If these enormous clinical differences occur, it is due to the patient and his or her immune status, not the virus. Therefore, it is urgent to study what determines this condition of the immune system. In addition, the clinical picture of severe Covid, characterised mainly by inflammatory pneumonia, appears after seven or eight days of flu symptoms, that is, when there are no longer active viruses in the body, despite the PCR test remaining positive (and this has been unequivocally demonstrated by viral culture) (2).
The inflammatory outbreak that occurred in the spring of 2020 is immunologically correlated with a syndrome of ADE or vaccine-augmented disease and it should be noted that ADE can also be produced or facilitated by the HA (hemagglutinin) antigen of influenza vaccines, which is has been shown to cross-react with the spike protein of coronaviruses (3). It is very significant that in 2019, new cell culture influenza vaccines were introduced, both in China and in Europe, containing theoretically non-replicating strains of coronavirus, as has been recognised by the WHO (4).
On the other hand, in various immunological studies, autoantibodies directed against different immune proteins have been found, including interferon (essential for the control of viral proliferation) which are much more frequent in people who develop severe Covid or what has come to be called “persistent Covid”.
In addition, the sustained production of antibodies against viral antigens that are compatible with antigens expressed by endogenous retroviruses, can lead to autoimmune phenomena or the maintenance of a chronic low-grade inflammatory profile that favors ADE or similar syndromes such as hemophagocytic syndrome, that is, a clinical profile of hyperinflammatory immunodeficiency.
In the autopsies of those deceased from Covid we see that the pathogenesis essentially affects the blood vessels, since severe Covid is basically an autoimmune vasculitis. This vasculitis can also be produced by the spike protein that the gene-based, so-called vaccines will force our cells to produce (5).
In conclusion, if the alleged Covid epidemic is triggered (not caused: a very relevant detail from an epidemiological point of view) by a hypothetical SARS-CoV-2 virus of which no official body has any culture or reference, It could have been ended a long time ago by simply allowing it to follow its natural course. But, as we will demonstrate in future presentations, it is being artificially maintained by gene-based drugs that are being called 'Covid vaccines'.
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References:
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Mechanisms of SARS-CoV-2 Evolution Revealing Vaccine-Resistant Mutations in Europe and America https://pubs.acs.org/doi/10.1021/acs.jpclett.1c03380
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CEBM Oxford, T. Jefferson et alt. ;https://www.medrxiv.org/content/10.1101/2020.08.04.20167932v4
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Non-neutralizing SARS CoV-2 directed polyclonal antibodies demonstrate cross-reactivity with the HA glycans of influenza virus. https://doi.org/10.1016/j.intimp.2021.108020
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Lombardia, cosa ti hanno fatto? Ecco risolto il mistero della Lombardia! By: Cristian Atzori Auret.
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SARS-CoV-2 Spike Protein Impairs Endothelial Function via Downregulation of ACE 2 https://www.ahajournals.org/doi/10.1161/CIRCRESAHA.121.318902
- Free SARS-CoV-2 Spike Protein S1 Particles May Play a Role in the Pathogenesis of COVID-19 Infection https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7772528/
via https://nomasmentiras.uy/2021/12/24/como-la-gripe-se-convirtio-en-covid/
Translation: David Montoute
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